Kidney Research and Clinical Practice

Review Article

March 23, 2021

T helper 17 cells in the pathophysiology of acute and chronic kidney disease

David P. Basile, Md Mahbub Ullah, Jason A. Collet, et al.

Kidney Res Clin Pract. 2021;40(1):12-28.

Both acute and chronic kidney disease have a strong underlying inflammatory component. This review focuses primarily on T helper 17 (Th17) cells as mediators of inflammation and their potential to...

Review Article

March 3, 2021

Pediatric acute kidney injury: new advances in the last decade

Sidharth K. Sethi, Timothy Bunchman, Ronith Chakraborty, et al.

Kidney Res Clin Pract. 2021;40(1):40-51.

Pediatric acute kidney injury (AKI) is a frequently missed complication. AKI has a significant impact on both short- and long-term outcomes in children. Within the last decade, there have been...

Original Article

March 5, 2021

Comparison of chronic kidney disease trial designs and analysis strategies

John Lawrence

Kidney Res Clin Pract. 2021;40(1):62-68.

Background: Despite the large burden of chronic kidney disease (CKD), it is challenging to conduct adequately powered clinical trials in this setting. Sound and efficient trials are needed to advance...

Original Article

March 12, 2021

Serum interferon-γ and urinary monocyte chemoattractant peptide-1 are important factors in the pathogenesis of immunoglobulin A nephropathy

Sang Youb Han, Kyung Hwan Jeong, Chun-Gyoo Ihm, et al.

Kidney Res Clin Pract. 2021;40(1):69-76.

Background: Imbalance of T helper (Th) 1/2 cells has been shown to contribute to the development of immunoglobulin A nephropathy (IgAN). To address the inconsistent results on the role of...

Original Article

March 24, 2021

Histopathologic and clinicopathologic classifications of antineutrophil cytoplasmic antibody-associated glomerulonephritis: a validation study in a Korean cohort

Jeong-Hoon Lim, Man-Hoon Han, Yong-Jin Kim, et al.

Kidney Res Clin Pract. 2021;40(1):77-88.

Background: Antineutrophil cytoplasmic antibodies (ANCA)-associated glomerulonephritis (AAGN) is a common cause of rapidly progressive glomerulonephritis and requires prompt and proper immunosuppressive therapy to improve renal prognosis. This study aimed to...

Original Article

March 22, 2021

The comparative effects of intravenous iron on oxidative stress and inflammation in patients with chronic kidney disease and iron deficiency: a randomized controlled pilot study

Xenophon Kassianides, Andrew Gordon, Roger Sturmey, et al.

Kidney Res Clin Pract. 2021;40(1):89-98.

Background: Concerns exist regarding the pro-oxidant and inflammatory potential of intravenous (IV) iron due to labile plasma iron (LPI) generation. This IRON-CKD trial compared the effects of different IV irons...

Original Article

March 22, 2021

Changes in plasma sclerostin level associated with use of a medium cut-off dialyzer in end-stage renal disease

Seon-Ho Ahn, Mi Mi Ko, Ju Hung Song, et al.

Kidney Res Clin Pract. 2021;40(1):120-134.

Background: Larger middle molecules are important substances associated with cardiovascular complications in end- stage renal disease. Unfortunately, larger middle molecules are not reliably removed by a high-flux dialyzer. A medium...

Original Article

March 5, 2021

Impact of acute kidney injury in deceased donors with high Kidney Donor Profile Index on posttransplant clinical outcomes: a multicenter cohort study

Woo Yeong Park, Yoon Kyung Chang, Young Soo Kim, et al.

Kidney Res Clin Pract. 2021;40(1):162-174.

Background: This study evaluated the impact of acute kidney injury (AKI) on posttransplant clinical outcomes for deceased donor (DD) kidney transplantation (KT) using the Kidney Donor Profile Index (KDPI) system....

Current Issue: Volume 40(1); Mar 2021

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Volume 40, No. 1

Kidney Research
and
Clinical Practice

Print ISSN: 2211-9132
Online ISSN: 2211-9140

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622	Cumulative fluid balance and mortality in elderly patients with acute kidney injury requiring continuous renal-replacement therapy: a multicenter prospective cohort study
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Cited By 28	The role of oxidative stress and hypoxia in renal disease
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